Histological changes in the lung in diseases associated with pulmonary venous hypertension
References (17)
- et al.
Amer. Heart J.
(1956) - et al.
Dis. Chest
(1957) - et al.
J. Fac. Radiol.
(1955) Bull. Johns Hopkins Hosp.
(1957)- et al.
Circulation
(1951) - et al.
J. Clin. Invest.
(1950) - et al.
Amer. med. Ass. Arch. Int. Med.
(1951) - et al.
J. Path. Bact.
(1958)
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Design of the β3-Adrenergic Agonist Treatment in Chronic Pulmonary Hypertension Secondary to Heart Failure Trial
2020, JACC: Basic to Translational ScienceCitation Excerpt :Although IpcPH is entirely reversible when pulmonary arterial wedge pressure is normalized by medical or surgical interventions, this is not the case for CpcPH, due to associated pulmonary arterial remodeling. The latter correlates with pulmonary vascular gradients and increased PVR (49). Approximately 12% to 14% of patients with PH and HF present with CpcPH, a prevalence similarly distributed in reduced or preserved HF (6).
Clinical Relevance of Computed Tomography Pulmonary Venography
2016, Heart Lung and CirculationCitation Excerpt :Factors causing pulmonary venous stenosis are listed in Table 1. Typical symptoms and signs of pulmonary venous stenosis are shortness of breath, chronic cough, recurrent haemoptysis, pulmonary oedema, recurrent pleural effusions and pulmonary venous hypertension [3–7]. Common radiological findings are thickened interseptal lines, ground-glass opacity, enlarged hilar lymph nodes and pleural effusions.
Pulmonary hypertension and right ventricular dysfunction in left heart disease (group 2 pulmonary hypertension)
2012, Progress in Cardiovascular DiseasesCitation Excerpt :Smooth muscle growth and migration are activated through serine elastase, growth factors and glycoproteins (tenascin-C and fibronectin).30-33 This is followed by arterialization of pulmonary veins.34 The vasculature's impaired response to chronic pressure elevation is also mediated by the endothelium.
Histopathology of pulmonary hypertensive diseases
2006, Current Diagnostic PathologyIs the left atrial v wave the determinant of peak pulmonary artery pressure in patients with pure mitral stenosis?
2000, American Journal of CardiologyCitation Excerpt :Vasoactive substances such as endothelin8 and adrenomedullin9 have been linked to the development of pulmonary hypertension. It may also occur from true anatomic remodeling, i.e., medial hypertrophy and intimal thickening.10 It is generally thought that the severity of MS and the resultant increase in LA pressure are important factors affecting the development of secondary pulmonary hypertension.
Alteration of the alveolar-capillary membrane diffusing capacity in chronic left heart disease
1998, American Journal of Cardiology
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Dr. Heath was in receipt of a Rockefeller Travelling Fellowship in Medicine while participating in this investigation, which was supported in part by research grant No. H3531 from the National Heart Institute, National Institutes of Health, United States Public Health Service.