ReviewGenetic factors in the aetiology of malignant mesothelioma
References (67)
- et al.
A genetic model for colorectal tumorigenesis
Cell
(1990) - et al.
Familial mesothelioma: a report of two families
Hum Pathol
(1989) An epidemiological survey of immunological abnormalities in asbestos workers. I. Non-organ and organ specific autoantibodies
Environ Res
(1980)- et al.
Diploid predominance and prognostic significance of S-phase cells in malignant mesothelioma
EurJ Cancer
(1991) - et al.
Epithelioid pleural mesotheliomas and pulmonary adenocarcinomas; a comparative DNA flow cytometric study
Hum Pathol
(1991) - et al.
Comparison of DNA and karyotype ploidy in malignant mesothelioma
Cancer Genet Cytogenet
(1992) - et al.
Malignant mesothelioma: clinical characteristics, asbestos minerology and chromosomal abnormalites in 41 patients
EurJ Cancer
(1992) - et al.
Cytogenetic anlaysis of malignant mesothelioma
Cancer Genet Cytogenet
(1990) - et al.
A genetic model for colorectal tumorigenesis
Cell
(1990) Health implications of environmental exposure to asbestos
Environ Health Perspect
(1985)
Cancer Statisitcs Review 1973–1988
The incidence of malignant mesothelioma in Australia 1947–1980
Med J Aust
Trends in mesothelioma incidence in Norway
Ann NY Acad Sci
Diffuse pleural mesothelioma in the Northwestern Cape Province
Br J Ind Med
Changing risk groups for malignant mesothelioma
Cancer
Asbestos associated chromosomal changes in human mesothelial cells
Familial mesothelioma; review and family study
Cancer Genet Cytogenet
Mesothelioma of pleura and peritoneum following exposure to asbestos in the London area
Br J Ind Med
Household contact asbestos neoplastic risk
Ann NY Acad Sci
Non-occupational exposure to asbestos and malignant mesothelioma in females
Lancet
Familial mesothelioma after intense asbestos exposure at home
JAMA
Familial clustering of malignant mesothelioma
Cancer
Malignant mesothelioma: clustering in a family producing asbestos cement in their home
Br J Ind Med
Malignant mesothelioma in two pairs of siblings; is there a hereditary predisposing factor
EurJ Resp Dis
Immune function related to asbestos exposure and mesothelioma, and immunotherapy for mesothelioma
Humoral immunologie abnormalities in workers exposed to asbestos cement dust
J Allergy Clin Immunol
Immunologie investigations in asbestos exposed workers
Chest
High frequency of immune dysfunction in asbestos workers and in patients with malignant mesothelima
J Clin Immunol
Rheumatoid factor in serum of individuals exposed to asbestos
Ann NY Acad Sci
The HLA system in asbestos workers
Br J Int Med
Clinical research on the treatment of locally advanced lung cancer
Radiotherapy plus 5-FU compared to radiotherapy alone for inoperable and unresectable bronchogenic carcinoma
Cancer
Cited by (48)
Malignant and Borderline Mesothelial Tumors of the Pleura
2018, Practical Pulmonary Pathology: A Diagnostic Approach A Volume in the Pattern Recognition SeriesRisk of second primary cancers after malignant mesothelioma and vice versa
2016, Cancer LettersCitation Excerpt :Nevertheless, on average only 10% of the people exposed to high levels of asbestos develop MM and a substantial minority of MM patients without known exposure have also been reported [3,10], suggesting possible involvement of other risk factors [11]. Genetic susceptibility may play a role in its etiology and familial clustering of MM has been reported [3,11–13]. Multiple or second primary cancers (SPCs) have been proposed to be a possible indicator for genetic or familial clustering of cancers [14].
Malignant and Borderline Mesothelial Tumors of the Pleura
2011, Practical Pulmonary Pathology E-Book: A Diagnostic Approach, Second EditionMalignant and Borderline Mesothelial Tumors of the Pleura
2011, Practical Pulmonary Pathology: A Diagnostic Approach A Volume in the Pattern Recognition SeriesMalignant mesothelioma of the tunica vaginalis testis: A case report and literature review
2009, Kaohsiung Journal of Medical SciencesCytogenetic and molecular genetic changes in malignant mesothelioma
2006, Cancer Genetics and CytogeneticsCitation Excerpt :It has been suggested that the variable incidence of SV40 sequences shown in MM may relate to the geographic differences in the incidence of MM or may reflect the problematic nature of the SV40 studies concerning the polymerase chain reaction (PCR) technique [reviewed in 16,17]. The latency period between the first exposure to asbestos and the diagnosis of MM ranges from 20 to 40 years, suggesting that multiple genetic alterations are needed for malignant transformation of the mesothelial cells [18]. A large number of cytogenetic studies have shown the complex nature of these changes, and numerous molecular genetic studies have revealed a number of genes with mutations or aberrant expression.