Elsevier

American Journal of Otolaryngology

Volume 23, Issue 6, November–December 2002, Pages 386-389
American Journal of Otolaryngology

Case Reports
Adult laryngomalacia: An uncommon clinical entity*,**,

Presented at the Virginia Society of Otolaryngology Head and Neck Surgery Annual Meeting, April 28, 2000, Hot Springs, VA.
https://doi.org/10.1053/ajot.2002.126322Get rights and content

Abstract

A 27-year-old female presented with a several-day history of acute onset inspiratory stridor and shortness of breath that worsened with phonation and minimal exertion. Flexible fiberoptic direct laryngoscopy revealed prolapse of the mucosa overlying the arytenoid cartilages bilaterally, consistent with type 1 laryngomalacia. These symptoms persisted with only minimal improvement despite administration of short-term corticosteroids, several weeks of antireflux medications, and other conservative measures. The patient underwent a supraglottoplasty and exhibited a marked improvement in her symptoms. The literature describes several cases of exercise-induced laryngomalacia in both pediatric and adult populations in which symptoms of inspiratory stridor and shortness of breath are induced by exercise but resolve upon its discontinuation. Adult laryngomalacia appears to be a clinical entity distinct from exercise-induced laryngomalacia because symptoms fail to resolve after several weeks of medical therapy and discontinuation of exertional activity. This case suggests that adult laryngomalacia, unlike pediatric and exercise-induced laryngomalacia, is less likely to resolve over time with conservative management and may require surgical intervention with supraglottoplasty necessary to alleviate symptoms. (This is a US government work. There are no restrictions on its use.)

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Discussion

Laryngomalacia can be divided into 3 broad categories—congenital, acquired, and exercise-induced. Gastroesophageal reflux (GER) has been indentified in childhood laryngeal anomalies, and congenital laryngomalacia is recognized as the most common cause of inspiratory stridor in infants. The natural history of this disorder is complete resolution in most children by age 12 to 18 months.1 The etiology, although unknown, is thought to be secondary to laryngeal “hypotonia” or poor neurologic

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*

Dr. Greinwald is currently affiliated with Department of Otolaryngology, Children's Medical Center, Cincinnati, OH.

**

The views expressed in this article are those of the authors and do not reflect the official policy or position of the Department of the Navy, Department of Defense, or the United States Government.

This is a US government work. There are no restrictions on its use.

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